To validate the role of ALKBH5 in modulating the m6A modification of MYO1C, we performed MeRIP-seq in ALKBH5-knockdown RA FLSs compared with RA FLSs transfected with the vector control and observed a hyper-m6A-peaks for MYO1C upon ALKBH5 silencing (Fig. 6H). This evidence concerns the gene ALKBH5 and rheumatoid arthritis.