VIM and Hepatic fibrosis: At 7 dpci, the prominence of fibrosis and tissue remodeling in the damaged zone were highlighted by the presence of the “Hepatic fibrosis/Hepatic stellate cell activation” as the most significant pathway (Fig. 1e), and by the higher accumulation of both vimentin and smooth muscle actin (Supplementary Figs. 4e, f and 5), reported to be markers for post-MI fibroblasts in zebrafish36.