It seems to promote glycolytic energy production by upregulating glucose transporters’ expression (i.e. GLUT1 and GLUT3) and transcription of enzymes responsible for intracellular glucose breakdown through glycolysis (e.g. Hexokinase, Phosphofructokinase-1, Aldolase), similarly to observations in Cancer-Associated Fibroblasts (CAFs) [77,78]. This evidence concerns the gene SLC2A3 and cancer.