CXCL1 and neoplasm: Similarly, miR-425-5p mimics further strengthened the expression intensity of TGFβ1, α-SMA, vimentin, Ki67, and CXCL1 in tumor tissues of mice injected with CAFs derived from MDA-MB-231 cells, while miR-425-5p inhibitors suppressed the expression intensity of TGFβ1, α-SMA, vimentin, Ki67, and CXCL1 to the greatest extent possible in tumor tissues (Figure 6(h)).