The results showed that the risk of death of high-risk patients (n=96) was significantly higher than that of patients with low risk scores (n=117), and our model could better distinguish the distribution of 4 genes expression in the cohort of low-risk and high-risk PCa patients, among which FCGR3A was the best and CCR7 and CD28 were the worst (Figure 2A). This evidence concerns the gene CD28 and posterior cortical atrophy.