Among them, CD3D, CD247, CD96 and G Protein-Coupled Receptor 18 (GPR18) were found to be relatively overexpressed in normal blood samples and BMX Non-Receptor Tyrosine Kinase (BMX), Mitogen-Activated Protein Kinase 14 (MAPK14), Complement C3b/C4b Receptor 1 (CR1), and C-Type Lectin Domain Family 4 Member D (CLEC4D) were found to be upregulated in sepsis samples (Figure 2F). This evidence concerns the gene BMX and Sepsis.