This may be potentially significant for the expression and potentiation of PD-related pathology as protein glycosylation regulated by B4Galt1 has been suggested to be related to microglial activation and neuroinflammatory responses (Yang et al., 2020), with increased expression of B4Galt1 related to increased microglial inflammatory responses (Yang et al., 2020). This evidence concerns the gene B4GALT1 and Parkinson disease.