FOXM1 has been reported to upregulate BRIP1 [38] and NBS1 [39], and FOXM1 activation upregulates RAD51 and BRCA1 in idiopathic pulmonary fibroblasts to activate HRR [40], with FOXM1 being a transcription factor that plays an important role in proliferation, cell cycle control, DNA repair, tumorigenesis, cancer progression, and tumor growth [41,42]. The gene discussed is BRIP1; the disease is neoplasm.