HSP90AA1 and breast cancer: Some enzymes involved in HALLMARK_FATTY_ACID_METABOLISM, also upregulated in overexpressed JTB condition, were found as upregulated, such as ENO2, above mentioned as a glycolytic enzyme that also contributes to the increased fatty acid production, and HSP90AA1 that also may activate MTORC1 signaling pathway, usually overexpressed in BC cells.