Of these more abundant proteins in the asbestos-exposed group, three were validated by Western blot analysis, all of which were acute-phase proteins: haptoglobin; ceruloplasmin, the copper carrying glycoprotein previously seen to be increased in MM patients’ blood and asbestos exposed individuals [43]; and fibulin-1, a member of the fibulin family, of which, fibulin-3 has been suggested as being implicative of asbestos exposure and MM [44]. The gene discussed is HP; the disease is Miyoshi myopathy.