Since day 1, the expression of kidney stress biomarkers was significantly higher in the AKI group, namely [TIMP2] × [IGFBP-7] 0.16 (ng/mL)2/1000 (IQR: 0.05–0.32) in the AKI group vs. 0.07 (ng/mL)2/1000 (IQR: 0.04–0.11) in the non-AKI group (p = 0.045); and NGAL 54.7 ng/mL (IQR: 36.4–117.4) in the AKI group vs. 32.4 (IQR: 14–40.05) in the non-AKI group (p = 0.002). The gene discussed is TIMP2; the disease is acute kidney injury.