Similarly, the ejection fraction showed a preserved cardiac function in CCR1−/− (46.3 ± 4.6%) and CCR2−/− (43.3 ± 2.7%) mice but not in CCR5−/− (32.7 ± 6.6%) and CX3CR1−/− (28.4 ± 4.5%) mice after myocardial infarction induction compared to wild-type mice (35.2 ± 4.7%, Figure 2B), as assessed by echocardiography. This evidence concerns the gene CX3CR1 and myocardial infarction.