We performed myocardial infarction in CCR1−/− (reduced neutrophils recruitment [25]) mice, CCR2−/− (reduced inflammatory monocyte recruitment [28,29]) mice, CCR5−/− (reduced T-cell recruitment [31]) mice and CX3CR1−/− mice (reduced reparatory monocyte recruitment [32]) mice. This evidence concerns the gene CCR2 and myocardial infarction.