Among the molecules activated by STING signaling, IFNs can stimulate the generation of anti-tumor T cells, T-cell infiltration and the direct killing of cancer cells [20,21,22], whereas CXCL10 and CCL5 are important for recruiting tumor-reactive effector T cells [14,15,16,23,24,25]. The gene discussed is STING1; the disease is neoplasm.