The pathway and gene network analyses revealed significant alterations in the PPAR, IL6, IL10, FOXO, JAK/STAT and NF-κB signaling pathways and potentially critical roles of IL33, VEGF, INSR, FOS, TGf-β, LIFR, and immunoglobulin that may be relevant to the pathophysiology of OHSS [52,53,54]. This evidence concerns the gene SOAT1 and ovarian hyperstimulation syndrome.