We previously showed in suspension cells (Jurkat T cells) and adherent monolayer culture (HT-29 colon carcinoma cells) that MTO loaded onto SPIONs induced the same cell death phenotype as its soluble counterpart, but that the velocity of cell death induction was slightly delayed [13,16]: this may have been due to a slower cellular uptake of the particles, compared to soluble drugs. Here, SELENBP1 is linked to colon carcinoma.