The analysis of the transposon integration sites identified several candidate genes, of which Man1a1, Pkp4, Rab10, Rasa1, Trps1, Vps26a, Xpnpep3 and Znf326 accelerated tumor growth, whereas the silencing of R3hcc1l reduced tumor growth. This evidence concerns the gene XPNPEP3 and neoplasm.