Later, in 2016, the results from a study showed that the complex of quercetin (25) and TNFRSF10A triggers apoptosis in breast cancer cells, as indicated by increased PARP cleavage and activation of the executioner caspases; CASP3 and CASP7, which have therapeutic potential for all types of hormone-dependent and triple-negative breast cancer (TNBC) cells [276]. The gene discussed is PARP1; the disease is breast carcinoma.