Similarly, Wang et al. found that YAP played a regulatory role in the process of melatonin maintaining the stemness of BMMSCs25; Wang et al. demonstrated that BMMSC‐derived extracellular vesicles induced cartilage reconstruction in temporomandibular joint OA through the autotaxin‐YAP signalling axis,6 while Mao et al. found that BMMSC‐derived exosomes regulated the GPRC5A‐YAP signalling axis to improve sulfur mustard‐induced acute lung injury.32 The gene discussed is GPRC5A; the disease is injury.