Using an m6A-circRNA epitranscriptomic chip, Wang et al. found that the screened hsa_circ_0000672 and hsa_circ_0005654 were explicitly participated in silica-induced pulmonary fibrosis by targeting eukaryotic translation initiation factor 4A3 (EIF4A3), suggesting that m6A RNA methylation of circRNAs mediated silica-induced fibrosis [34]. Here, EIF4A3 is linked to pulmonary fibrosis.