Furthermore, it was determined that α-DG was hypoglycosylated both in AML patients and the two cell lines (Tables 1, 2), which was associated with a downregulation of POMT1, POMT2, POMGNT1, FKTN, FKRP and LARGE1 genes in the two cell lines (Table 4), and either upregulation or downregulation of these genes in cells of patients (Table 3), as mentioned above. The gene discussed is POMT2; the disease is acute myeloid leukemia.