As previous experiments were performed in CD34+ cells, and in order to prove that the observed effect on erythropoiesis could be driven by the DDIT3 upregulation observed in HSCs from MDS patients, we also induced DDIT3-overexpression in primary HSCs isolated from healthy donors (YH_27, YH_28, YH_29). Here, DDIT3 is linked to myelodysplastic syndrome.