Besides, the rapid initiation of CXCL10 and IL-2, and the resultant hyper-expression of IL-6 with a coexisting deficiency in IL-10 expression are believed to add to the immuno-pathological activities related to pulmonary impairment in the course of SARS-CoV infection (Chien et al., 2006[27]). Here, CXCL10 is linked to severe acute respiratory syndrome.