ZIKV infection was associated with increased secretion of various chemokines (CCL5, CXCL1, CXCL8, CXCL10, and CXCL2) directly linked to the recruitment of immune cells, including T cells, macrophages, and NK cells that are known to actively contribute to the uterine vascular remodeling and the maintenance of fetal tolerance. The gene discussed is CXCL10; the disease is Zika virus infectious disease.