NFKB1 and early-onset autosomal dominant Alzheimer disease: The above-mentioned bacterial species are B. adolescentis, a bacterium with the capacity to sustain the activation of nuclear factor-κB (NF-kB), F. prausnitzii, that is able to increase the expression of the anti-inflammatory cytokine IL-10, through induction of human colonic regulatory T cells, E. rectale, a bacterium that has been already associated with a diminution of inflammation in Alzheimer’s disease, Ruminococcus obeum and D. formicigenerans, also known by their anti-inflammatory properties (Zuo et al., 2020a).