RPL10 and acute lymphoblastic leukemia: Given the known impact of R98S in T-ALL cells in terms of cellular proliferation, ribosome biogenesis, cell metabolism and resistance to pro-apoptotic stimuli (de Keersmaecker et al., 2013; Girardi et al., 2018; Kampen et al., 2019a; Kampen et al., 2019b), and based on our modelling of the Q123R RPL10 structure (Figure 4), we posited that the Q123R mutation could also have a role in T-ALL.