Besides, viral particles upregulated TLR3 and TSLP expression as previously described in dendritic cells, where these molecules promote the differentiation of Th17 cells under Th2 polarizing conditions24, and in infants with bronchiolitis where this Th2 environment can lead to asthma and allergic diseases in the long term47. The gene discussed is TLR3; the disease is bronchiolitis.