Within the lung microenvironment, γδ T cells are activated to produce IL-17A by tumor-derived or microbiota-induced IL-1β and IL-23 (Coffelt et al., 2015; Jin et al., 2019; Wellenstein et al., 2019), two cytokines with well-established influence on IL-17A–producing γδ T cells (Sutton et al., 2009). The gene discussed is IL17A; the disease is neoplasm.