Under the chemotactic effect of CXCL9, CXCL10, and CXCL11, activated Th1 cells reach the tumor site to exert phagocytosis and upregulate CXCL9, CXCL10, and CXCL11 secretion, thus recruiting more T lymphocytes to participate in the immune response at the lesion site. The gene discussed is CXCL11; the disease is neoplasm.