For example, the blockade of H4 receptors displayed antipruritic and anti‐inflammatory effects in atopic dermatitis,31, 32 possibly through the inhibition of both direct sensory nerve stimulation and increased IL‐31 and thymic stromal lymphopoetin (TSLP) production by inflammatory cells.33, 34, 35. The gene discussed is TSLP; the disease is atopic eczema.