In agreement with the results obtained in EphA2 KO cells pointing out that EphA2 is not the principal progranulin receptor in mesothelioma, in phospho-RTK arrays-based experiments we did not detect any increase in EphA2 tyrosine phosphorylation in mesothelioma cells stimulated with progranulin, indicating that EphA2 is not directly activated by progranulin in this model. The gene discussed is EPHA2; the disease is mesothelioma.