Inhibition of gga-miR-365-3p and overexpression of SOCS5 reduced cellular secretion of pro-inflammatory factors TNF-α and IL-6 and boosted intracellular pMGA1.2 expression, which resulted in reducing cellular inflammatory damage while also facilitating MG survival (Figures 2 and 8). The gene discussed is IL6; the disease is myasthenia gravis.