An incidental finding was that not only interleukin-17A was above average, but interleukin-17C, which is a significant cytokine in the signaling pathway of colorectal cancer, promotes development by activating anti-apoptosis markers- B-cell lymphoma (Bcl-2) and B-cell lymphoma- extra-large (Bcl-xL) was also noticed to be increased in colorectal cancer patients when studied in both humans and animal model (mouse) [13]. This evidence concerns the gene BCL2 and colorectal cancer.