The data from the Beat AML leukemia dataset show that a number of tumor samples with cytogenetic deletions of all or a part of 20q had high levels of expression of SGK2 or L3MBTL1 (Additional file 12: Fig. S4), suggesting possible compensatory mechanisms of upregulation of expression of the remaining allele of both genes or potential tumor heterogeneity of some samples. This evidence concerns the gene L3MBTL1 and acute myeloid leukemia.