The data from the Beat AML leukemia dataset show that a number of tumor samples with cytogenetic deletions of all or a part of 20q had high levels of expression of SGK2 or L3MBTL1 (Additional file 12: Fig. S4), suggesting possible compensatory mechanisms of upregulation of expression of the remaining allele of both genes or potential tumor heterogeneity of some samples. Here, SGK2 is linked to acute myeloid leukemia.