Despite antibiotic resistance being the primary driver of clinical outcome in A. baumannii infections, the virulence properties such as capsular lipopolysaccharide presence and biofilm formation, effectively trigger lipopolysaccharide (LPS)–Toll-like receptor 4 (TLR4)-mediated sepsis by reaching high bacterial density in a short time span2. This evidence concerns the gene TLR4 and Sepsis.