Molecular docking analysis showed that quercetin, luteolin, kaempferol, stigmasterol, and sitosterol, can interact with CDKN1A, CDK1, MYC, PLAU and MCL1, etc. Previous studies have reported that quercetin-induced growth inhibition and cell death in prostatic carcinoma cells are associated with increased CDKN1A levels (24). This evidence concerns the gene CDK1 and prostate carcinoma.