Yi et al.104found that the novel RNA‐binding protein polymerase I and transcript release factor (PTRF) maintained the mRNA stability of lncRNA NEAT1 and inhibited UBX domain protein 1 (UBXN1) expression, consequently activating the NF‐κB signalling pathway, promoting the binding of NF‐κB to the PD‐L1 promoter region, and enhancing the transcription of PD‐L1, ultimately promoting the immune evasion of GBM cells. The gene discussed is UBXN1; the disease is glioblastoma.