Wei et al.127also found that EIF4A3‐induced circASAP1 (ArfGAP with SH3 domain, ankyrin repeat and PH domain 1) was markedly upregulated in recurrent GBM tissues and temozolomide‐resistant cell lines, and circASAP1 upregulated NRAS expression by sponging miR‐502‐5p, increasing the proliferation of temozolomide‐resistant GBM cells and their resistance to temozolomide. The gene discussed is NRAS; the disease is glioblastoma.