NFKB1 and glioblastoma: Yi et al.104found that the novel RNA‐binding protein polymerase I and transcript release factor (PTRF) maintained the mRNA stability of lncRNA NEAT1 and inhibited UBX domain protein 1 (UBXN1) expression, consequently activating the NF‐κB signalling pathway, promoting the binding of NF‐κB to the PD‐L1 promoter region, and enhancing the transcription of PD‐L1, ultimately promoting the immune evasion of GBM cells.