A study using rat models of Parkinson’s disease (PD) established through the injection of 6-hydroxydopamine revealed that the downregulation of lncRNA BACE1-AS inhibited inducible nitric oxide synthase, α-synuclein, and glutamic acid activation and elevated dopamine and TH levels to improve oxidative stress injury in rats with PD by upregulating miR-34b-5p via integrating with it and indirectly downregulating BACE1 [90]. The gene discussed is TH; the disease is Parkinson disease.