It was inferred that NEAT1 acted as a competing endogenous RNA, regulating the miR‐34b‐5p–GLI1 axis through declining the miR‐34b‐5p level and increasing GLI1 level, further accelerating the proliferation of DLBCL cells, which was often associated with disease progression and poor prognosis (Fig. 2D) [52]. This evidence concerns the gene GLI1 and diffuse large B-cell lymphoma.