Previously described genes DACH1, MYH10, NOTCH2, TBX4, EVC2, OTOG, and SHC3 have highly damaging variants also in our study group; of these TBX4, EVC2, NWD1, and OTOG have variants in more than one family further strengthening the notion that they are involved in the etiopathogenesis of DDH. This evidence concerns the gene SHC3 and Hip dysplasia.