Neutrophil CXCR2 is essential for its emigration from bone marrow and trafficking toward sites of inflammation and tumors.[10] As expected, intraperitoneal injection of the CXCR2 antagonist SB225002 in Setd2KO tumor‐bearing mice significantly decreased tumor burden in the pancreas, further reinforcing the protumoral function of neutrophils in Setd2KO tumors (Figure 2B,C; Figure S4A, Supporting Information). The gene discussed is CXCR2; the disease is neoplasm.