The results of functional enrichment analysis showed that PL played a pharmacological role in the treatment of PCa by regulating the metabolic processes of reactive oxygen species, response to steroid hormones, and oxidative stress as well as IL-17 signaling pathway, PCa, and estrogen signaling pathway, which involved biological processes such as sensitivity to steroid hormones, cellular metabolism, cytokines, and gene transcription. The gene discussed is IL17A; the disease is posterior cortical atrophy.