AR and neoplasm: Tumor and normal ranked ARBS rankings follow strikingly similar distributions considering the genetic heterogeneity of tumors10,11, suggesting that AR enhancer heterogeneity is not tumor-intrinsic, but instead patient-intrinsic (Fig. 1d, Supplementary Fig. 1G), corroborating our previous case-study identifying high inter-metastatic AR binding overlap from the same patient7.