Increased deposition of ApoB-containing lipoproteins in muscle, such as LDL-C, can also increase inflammatory cell recruitment, which was a key observation in our dyslipidemic models of DMD and LGMD2B [11, 23], and potentially rationalizes the misdiagnosis of LGMD2B for polymyositis. Here, APOB is linked to Duchenne muscular dystrophy.