For example, Serum amyloid A (SAA), as the main component of the acute-phase inflammatory response, has been shown to be more sensitive to cardiovascular and non-cardiovascular events than CRP, and patients with elevated SAA levels have a higher incidence of adverse events (mortality, recurrent angina, acute myocardial infarction, stroke, and other vascular events) [9, 10]. This evidence concerns the gene SAA1 and stroke disorder.