IL17A and hyperinsulinemic hypoglycemia, familial, 4: In this study we also found that the peak of some proinflammatory cytokines (γ‐IFN and IL‐17A) released mainly by T cells were not as high as those secreted by innate immune cells in patients with extreme deficiency of the adaptive immune system after HIV infection, although γ‐IFN deficiency increased T. marneffei infection and proliferation.25