To better assess the relationship between Sox9 and the phenotypes observed following LATS1/2-deletion, we compared gene expression signatures from our LATS1/2 deletion model to those from a previous study that compared gene expression of mammary epithelial cells with high Sox9 levels (Sox9hi) and low Sox9 levels (Sox9lo) in the C3(1)-Tag basal-like breast cancer mouse model7. This evidence concerns the gene SOX9 and breast carcinoma.