GSEA with our LATS-null ranklist displayed a strong enrichment for upregulated genes in the Brca1f/f; p53f/+; Blg-Cre model, suggesting a correlation between LATS1/2 inactivation and basal-like carcinomas driven by the Brca1 mutation (Supplementary Fig. 4c). This evidence concerns the gene BRCA1 and carcinoma.