KRT10 and neoplasm: The mutations observed here were evaluated as genetic markers: whether K5-K6 region had read over-coverage over 1.5X, whether K8.1 gene was inactivated, whether miR-K10 had point mutations relative to database consensus, whether K4.2 was truncated, whether K11.2 174 bp central domain was duplicated (Fig 8, S7 Table), whether any of these mutations significantly occurred together (S8 Table), and whether these mutations were associated with any clinical or individual tumor traits (S7 and S9 Tables).