Beyoğlu et al. classified HCC patients into six subgroups according to the presence of HBV infection, p53 mutation, and activation of Wnt/β-catenin signaling, and they found that HCC with HBV infection resulted in markedly reduced tissue concentrations of 1-stearoylglycerol, 1-palmitoylglycerol, and palmitic acid [32]. The gene discussed is TP53; the disease is hepatocellular carcinoma.