Yu et al. found that levels of lTSLP were significantly increased in asthma airway epithelial cells promoting miRNA-233/Von Hippel-Lindau (VHL)/HIF-1α-dependent pro-inflammatory cytokine production and aerobic glycolysis from airway epithelial cells, whereas levels of sTSLP were decreased (Fig. 2). This evidence concerns the gene HIF1A and asthma.