These results are in accordance with those from our previous study on the pathogenesis of BPD, which suggested that cellular lipid membrane activities, such as those of the endoplasmic reticulum, were involved in the pathological process of BPD (39), and many studies also confirmed that pathways, such as YAP/TAZ mediate actin-dependent ATPase and endoplasmic reticulum function pathways (40–42), which are involved in the functional changes of alveolar epithelial cells. The gene discussed is DNAH8; the disease is bronchopulmonary dysplasia.