LITAF and hepatocellular carcinoma: One study on hepatocellular carcinoma exhibited the downregulation of CIDE‐C in HCC tissues compared with adjacent normal tissue, as well as an obvious proapoptotic effect caused by the overexpression of CIDE-C in HCC cells, which could be explained by the interaction between CIDE‐C and lipopolysaccharide-induced tumor necrosis factor (LITAF) 113.